Publikationen

  • Abdel-Fattah, W.; Scheidt, V.; Uthman, S.; Stark, M.; Schaffrath, R. (2013). Insights into Diphthamide, Key Diphtheria Toxin Effector. Toxins, 5(5), S. 958-968.
  • Woodacre, A.; Jablonowski, D.; Lone, M.; Schneiter, R.; Giorgini, F.; Schaffrath, R. (2013). A novel Sit4 phosphatase complex is involved in the response to ceramide stress in yeast. Oxidative Medicine and Cellular Longevity, 2013, S. 129645.
  • Eichler, C.; Herberg, F.; Schmidt, F.; Schaffrath, R. (2013). Does the bee locus encode an additional or alternative pilus in Enterococcus faecalis?. In: Mendez-Vilas A (Hrsg.), Worldwide Research Efforts in the Fighting against Microbial Pathogens: From Basic Research to Technological Developments, S. 134-138. . Boca Raton, FL, USA: BrownWalker Press.
  • Uthman, S.; Bär, C.; Liu, S.; ten Have, S.; Giorgini, F.; Stark, M.; Schaffrath, R.; Scheidt, V. (2013). The Amidation Step of Diphthamide Biosynthesis in Yeast Requires DPH6, a Gene Identified through Mining the DPH1-DPH5 Interaction Network. Plos Genetics, 9, S. e1003334.
  • Uthman, S.; Liu, S.; Giorgini, F.; Stark, M.; Costanzo, M.; Schaffrath, R. (2012). Diphtheria disease and genes involved in formation of diphthamide, key effector of the diphtheria toxin. In: Kumar R (Hrsg.), Insight and Control of Infectious Disease in Global Scenario, S. 333-356. . Rijeka, Croatia: INTECH Open Access Publisher.
  • Uthman, S.; Kheir, E.; Bär, C.; Jablonowski, D.; Schaffrath, R. (2011). Growth inhibition strategies based on antimicrobial microbes/toxins.. In: Mendez-Vilas A (Hrsg.), Science against Microbial Pathogens: Communicating Current Research and Technological Advances, S. 1321-1329. . Badajoz, Spain: Formatex Research Center.
  • Meineke, B.; Schwer, B.; Schaffrath, R.; Shuman, S. (2011). Determinants of eukaryal cell killing by the bacterial ribotoxin PrrC. Nucleic Acids Research, 39, S. 687-700. 
  • Kheir, E.; Bär, C.; Jablonowski, D.; Schaffrath, R. (2011). Cell growth control by tRNase ribotoxins from bacteria and yeast. In: Mendez-Vilas A (Hrsg.), Science and Technology against Microbial Pathogens. Research, Development and Evaluation, S. 398-402. . Singapore: World Scientific Publishing Co. Pte. Ltd.
  • Mehlgarten C, Jablonowski D, Wrackmeyer U, Tschitschmann S, Sondermann D, Jäger G, Gong Z, Byström AS, Schaffrath R, Breunig KD. (2010) Elongator function in tRNA wobble uridine modification is conserved between yeast and plants. Mol. Microbiol. 75, 1082-1094.2009:
  • Jablonowski D, Täubert J-E, Bär C, Stark MJR, Schaffrath R. (2009) Distinct subsets of Sit4 holo-phosphatases are required for inhibition of yeast growth by rapamycin and zymocin. Eukaryot. Cell 8, 1637-1647.
  • Mehlgarten C, Jablonowski D, Breunig KD, Stark MJR, Schaffrath R. (2009) Elongator function depends on antagonistic regulation by casein kinase Hrr25 and protein phosphatase Sit4. Mol. Microbiol., 73, 869-881.
  • Zabel, R., Bär, C., Mehlgarten, C., & Schaffrath, R. (2008). Yeast alpha-tubulin suppressor Ats1/Kti13 relates to the Elongator complex and interacts with Elongator partner protein Kti11. Mol. Microbiol., 69 , 175-187.
  • Bär, C., Zabel, R., Liu, S., Stark, M.J., & Schaffrath, R. (2008). A versatile partner of eukaryotic protein complexes that is involved in multiple biological processes: Kti11/Dph3. Mol. Microbiol., 69 , 1221-1233.
  • Studte, P., Zink, S., Jablonowski, D., Bär, C., von der Haar, T., Tuite, M.F., & Schaffrath, R. (2008). tRNA and protein methylase complexes mediate zymocin toxicity in yeast. Mol. Microbiol. 69, 1266-1277.
  • Nandakumar, J., Schwer, B., Schaffrath, R., & Shuman, S. (2008). RNA repair therapy: an antidote to cytotoxic eukaryal RNA damage. Mol. Cell, 31, 278-286.
  • Jablonowski, D., & Schaffrath, R. (2007). Zymocin, a composite chitinase and tRNase killer toxin from yeast. Biochem. Soc. Trans. 35, 1533-1537.
  • Chen, Z., Zhang, H., Jablonowski, D., Zhou, X., Ren, X., Hong, X., Schaffrath, R. , Zhu, J-K., & Gong, Z. (2006). Mutations in ABO1/ELO2 , one subunit of holo-Elongator, increase ABA sensitivity and drought tolerance in Arabidopsis . Mol. Cell. Biol. 26, 6902-6912.
  • Jablonowski, D., Zink, S., Mehlgarten, C., Daum, G., & Schaffrath, R. (2006). tRNA Glu wobble uridine methylation by Trm9 identifies Elongator's key role for zymocin-induced cell death in yeast. Mol. Microbiol. 59, 677-688.
  • Zink, S., Mehlgarten, C., Kitamoto, H.K., Nagase, J., Jablonowski, D., Dickson, R.C., Stark, M.J., & Schaffrath, R. (2005). M(IP) 2 C, the major yeast plasma membrane sphingolipid, governs toxicity of Kluyveromyces lactis zymocin . Eukaryot. Cell 4 , 879-889 .
  • Mehlgarten, C., & Schaffrath, R. (2004). After chitin-docking, toxicity of Kluyveromyces lactis zymocin requires Saccharomyces cerevisiae plasma membrane H + -ATPase. Cell. Microbiol. 6, 569-580.
  • Jablonowski, D., Fichtner, L., Stark, M.J., & Schaffrath, R. (2004). The yeast Elongator histone acetylase requires Sit4-dependent dephosphorylation for toxin-target capacity. Mol. Biol. Cell 15 , 1459-1469.
  • Fichtner, L., Jablonowski, D., Schierhorn, A., Kitamoto, H.K., Stark, M.J., & Schaffrath, R. (2003). Elongator's toxin-target ( TOT) function is NLS-dependent and suppressed by post-translational modification . Mol. Microbiol . 49, 1297-1307.
  • Frohloff, F., Fichtner, L., Jablonowski, D., Breunig, K.D., & Schaffrath, R. (2001). Saccharomyces cerevisiae Elongator mutations confer resistance to the Kluyveromyces lactis zymocin . EMBO J. 20 , 1993-2003.
  • Jablonowski, D., Butler, A.R., Fichtner, L., Gardiner, D., Schaffrath, R. , & Stark, M.J. (2001). Sit4p protein phosphatase is required for sensitivity of Saccharomyces cerevisiae to Kluyveromyces lactis zymocin . Genetics 159, 1479-1489.


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